Lab Studies:
CBC, CMP, TSH performed at initial work up were all within normal limits.
Other Studies:
CTA: no acute cardiopulmonary process. No evidence of PE.
<br>Holter monitor: 742 PVCs in 24 hours; 6 beats of NSVT (unclear morphology) vs SVT with aberrancy.
<br>Exercise Treadmill Stress ECG: No ischemic changes or dysrhythmias; PVCs from the inferior RV noted.
<br>Cardiac MRI:
<br> <a href="CaseStudyImages/CS1PalpitationsinaCo-396.jpg" title="Case Photo #1" data-lity><b>Case Photo #1</b></a> <a href="CaseStudyImages/CS2CS1Palpitationsin-396.jpg" title="Case Photo #2" data-lity><b>Case Photo #2</b></a> RV mildly enlarged. The diaphragmatic/inferior RV wall and RV apex are focally dyskinetic. There is delayed and hyperenhancement of the inferior and apical RV walls. This is compatible with fibrofatty infiltration of the RV.
<br>Signal Average ECG: Normal
<br>Genetic testing: Pathogenic mutation in PKP2 R79X (Plakophilin 2)
Consultations:
Cardiology
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